Part III of a conversation with a low-fat zealot and statin advocate

The low-fat zealot and statin advocate says,
"The mechanism is known. No speculation is required. Lipoproteins that carry the apo B (LDL and VLDL) adhere to the artery walls, other lipoproteins don't. The higher the concentration in the blood of apo B lipoproteins, the more they diffuse into the artery wall. Once inside the wall they become oxidized. Oxidized LDL stimulates inflammation. In response to the inflammation macrophages are attracted, they devour the oxidized LDL and send out more inflammatory chemicals. They become foam cells which grow and eventually burst leaving cholesterol crystals in the artery wall. The crystals penetrate the artery wall causing even more inflammation."

The model presented above is very appealing to those who believe that LDL cholesterol is "bad" and is the direct cause of atherosclerosis. But the model would have you believe that God/evolution sent LDL down to constantly war against the epithelium of arteries. Strangely enough LDL has declared peace with the epithelium of veins. Why is that? Also if concentration of LDL is so important why is the correlation of atherosclerosis with LDL levels so weak? Why do many people with hypercholesterolemia live a life untouched by atherosclerosis? This simplistic model serves the statin industry and their FDA captives quite will as long as it is not examined closely. I have to laugh every-time I see this model presented on TV by a statin advertiser.